Zentrum für translationale vaskuläre Biologie (CTVB)
Forschungsschwerpunkt / Johannes Gutenberg-Universität Mainz
Barco, Stefano
Edoxaban for pulmonary embolism with right ventricular dysfunctionLancet Haematology. Bd. 3. H. 9. 2016 S. E405-E406
Jimenez, David; Luis Lobo, Jose; Fernandez-Golfin, Covadonga et al.
Effectiveness of prognosticating pulmonary embolism using the ESC algorithm and the Bova scoreTHROMBOSIS AND HAEMOSTASIS. Bd. 115. H. 4. 2016 S. 827-834
Brandt, M.; Garlapati, G.; Raaz, U. et al.
Effects of neurohormonal stimulation on cardiomyocyte telomere length and effects on cellular functionEUROPEAN HEART JOURNAL. Bd. 37. 2016 S. 435-435
Barco, S.; Barbar, S.; Kirschner, Y. et al.
Elevated levels of interleukin 6 at the time of acute pulmonary embolism diagnosis are associated with short- and long-term adverse outcomesEUROPEAN HEART JOURNAL. Bd. 37. 2016 S. 950-951
Ghiassian, Susan Dina; Menche, Jorg; Chasman, Daniel I. et al.
Endophenotype Network Models: Common Core of Complex DiseasesSCIENTIFIC REPORTS. Bd. 6. 2016
Bochenek, Magdalena L.; Schuetz, Eva; Schaefer, Katrin
Endothelial cell senescence and thrombosis: Ageing clotsTHROMBOSIS RESEARCH. Bd. 147. 2016 S. 36-45
Gogiraju, R.; Schroeter, M. R.; Bochenek, M. L. et al.
Endothelial deletion of protein tyrosine phosphatase-1B promotes angiogenesis and protects against pressure overload-induced heart failure in miceEUROPEAN JOURNAL OF HEART FAILURE. Bd. 18. 2016 S. 164-164
Gogiraju, Rajinikanth; Schroeter, Marco R.; Bochenek, Magdalena L. et al.
Endothelial deletion of protein tyrosine phosphatase-1B protects against pressure overload-induced heart failure in miceCARDIOVASCULAR RESEARCH. Bd. 111. H. 3. 2016 S. 204-216
Gogiraju, R.; Hubert, A.; Muenzel, T. et al.
Endothelial leptin receptor signalling contributes to the development of cardiac hypertrophy and fibrosis following chronic pressure overload in miceEUROPEAN JOURNAL OF HEART FAILURE. Bd. 18. 2016 S. 132-132
Kroeller-Schoen, S.; Jansen, T.; Tran, Y. et al.
Endothelial PGC-1alpha preserves endothelial function during chronic angiotensin II treatment by maintaining eNOS expression, reducing vascular oxidative stress and supporting the antioxidant defenseEUROPEAN HEART JOURNAL. Bd. 37. 2016 S. 812-812