Muellerleile, Kai; Groth, Michael; Steven, Daniel et al.
Cardiovascular Magnetic Resonance Demonstrates Reversible Atrial Dysfunction After Catheter Ablation of Persistent Atrial FibrillationJOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY. Bd. 24. H. 7. 2013 S. 762-767
Ariza, L.; Keller, K.; Schulz, A. et al.
Cardiovascular profile of individuals with deep vein thrombosis: results of the Gutenberg Health Study (GHS)JOURNAL OF THROMBOSIS AND HAEMOSTASIS. Bd. 11. 2013 S. 615-615
Keller, K.; Coldewey, M.; Ullmann, A. et al.
Cardiovascular risk profile in deep vein thrombosis and pulmonary embolism - Results from the thrombEVAL trialINTERNIST. Bd. 54. 2013 S. 77-78
Daub, Steffen; Hausding, Michael; Jurk, Kerstin et al.
CD40 ligand contributes to angiotensin-II induced vascular inflammation, oxidative stress and endothelial dysfunctionFASEB JOURNAL. Bd. 27. 2013
Hausding, Michael; Daub, Steffen; Jurk, Kerstin et al.
CD40 LIGAND PROMOTES ANGIOTENSIN-II INDUCED VASCULAR INFLAMMATION, THROMBIN SENSITIVITY, OXIDATIVE STRESS AND ENDOTHELIAL DYSFUNCTIONJOURNAL OF VASCULAR RESEARCH. Bd. 50. 2013 S. 29-29
Hausding, M.; Jurk, K.; Becker, C. et al.
CD40 ligand promotes angiotensin-II induced vascular inflammation, thrombin sensitivity, oxidative stress and endothelial dysfunctionEUROPEAN HEART JOURNAL. Bd. 34. 2013 S. 761-761
Hausding, Michael; Jurk, Kerstin; Daub, Steffen et al.
CD40L contributes to angiotensin II-induced pro-thrombotic state, vascular inflammation, oxidative stress and endothelial dysfunctionBasic research in cardiology. Bd. 108. H. 6. Heidelberg: Springer 2013 386
Jurk, K.; Hausding, M.; Muenzel, T. et al.
CD40L deficiency protects from a pro-thrombotic phenotype induced by angiotensin-II in miceJOURNAL OF THROMBOSIS AND HAEMOSTASIS. Bd. 11. 2013 S. 278-278
Espinola-Klein, Christine; Dopheide, Joern Fredrik; Gori, Tommaso
Change of ankle-brachial index and progression of atherosclerosisVASA-EUROPEAN JOURNAL OF VASCULAR MEDICINE. Bd. 42. H. 2. 2013 S. 86-87
Oelze, Matthias; Knorr, Maike; Kroeller-Schoen, Swenja et al.
Chronic therapy with isosorbide-5-mononitrate causes endothelial dysfunction, oxidative stress, and a marked increase in vascular endothelin-1 expressionEUROPEAN HEART JOURNAL. Bd. 34. H. 41. 2013 S. 3206-3216